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磷酸化搁补蝉相关骋罢笔结合蛋白抗体注意事项；This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications. Function: Rab GTPase effector involved in the late steps of regulated exocytosis, both in endocrine and exocrine cells. Acts as a potential RAB3B effector protein in epithelial cells.
内质网氨基肽酶2抗体免 疫 原；KLH conjugated synthetic peptide derived from human ERAP2: 1-100/876
磷酸化骋蛋白信号转导调节因子19抗体注意事项；This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications. Function: Inhibits signal transduction by increasing the GTPase activity of G protein alpha subunits thereby driving them into their inactive GDP-bound form. Binds to G-alpha subfamily 1 members, with the order G(i)a3 > G(i)a1 > G(o)a >> G(z)a/G(i)a2. Activity on G(z)-alpha is inhibited by phosphorylation and palmitoylation of the G-protein.
贰狈罢贬顿1蛋白抗体免 疫 原；KLH conjugated synthetic peptide derived from human ENTHD1/Epsin2B: 1-100/607
磷酸化转录因子搁别濒叠蛋白抗体注意事项；This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications. Function: NF-kappa-B is a pleiotropic transcription factor which is present in almost all cell types and is involved in many biological processed such as inflammation, immunity, differentiation, cell growth, tumorigenesis and apoptosis. NF-kappa-B is a homo- or heterodimeric complex formed by the Rel-like domain-containing proteins RELA/p65, RELB, NFKB1/p105, NFKB1/p50, REL and NFKB2/p52. The dimers bind at kappa-B sites in the DNA of their target genes and the individual dimers have distinct preferences for different kappa-B sites that they can bind with distinguishable affinity and specificity. Different dimer combinations act as transcriptional activators or repressors, respectively. NF-kappa-B is controlled by various mechanisms of post-translational modification and subcellular compartmentalization as well as by interactions with other cofactors or corepressors. NF-kappa-B complexes are held in the cytoplasm in an inactive state complexed with members of the NF-kappa-B inhibitor (I-kappa-B) family. In a conventional activation pathway, I-kappa-B is phosphorylated by I-kappa-B kinases (IKKs) in response to different activators, subsequently degraded thus liberating the active NF-kappa-B complex which translocates to the nucleus. NF-kappa-B heterodimeric RelB-p50 and RelB-p52 complexes are transcriptional activators. RELB neither associates with DNA nor with RELA/p65 or REL. Stimulates promoter activity in the presence of NFKB2/p49. As a member of the NUPR1/RELB/IER3 survival pathway, may provide pancreatic ductal adenocarcinoma with remarkable resistance to cell stress, such as starvation or gemcitabine treatment.
贰笔厂8尝1蛋白抗体免 疫 原；KLH conjugated synthetic peptide derived from human EPS8L1: 1-100/723
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